with DUPIXENT 300 mg Q2W + SOC (n=129) vs placebo + SOC (n=61) (rate ratio:
0.29 [95% CI:
0.17, 0.51]) (baseline blood EOS ≥300
cells/μL, QUEST, post hoc
analysis)2,4,5
Results are descriptive. Definitive conclusions cannot be made as this was a post-hoc
analysis. There are limitations on sample size and data
were not
multiplicity controlled.
eSevere exacerbations were defined as deterioration
of asthma requiring the use of
systemic corticosteroids for at least 3 days or hospitalization
or ED visit due to asthma that
required systemic corticosteroids.1
(Week 96) when treated with
DUPIXENT 300 mg Q2W + SOC
(n=215) for 52 weeks
in QUEST
and
continued with DUPIXENT
300
mg Q2W + SOC in the OLE
period
(baseline blood EOS ≥300
cells/μL,
TRAVERSE OLE study,
post hoc
analysis)2,4
Results are descriptive. Definitive conclusions cannot be made as this was a post hoc analysis of open-label extension data.
Data were not multiplicity controlled and there are limitations associated with
open-label study design, including lack of comparator arm, decreasing sample size, and
potential continued involvement of responders and attrition of
nonresponders.
Patients with higher short-acting beta agonist use (3+ canisters/
year)
were more likely to
experience 1+ severe exacerbation requiring an
HCP visit or
hospitalization6,f
fBased on observational analyses of SABA use IN Asthma
(SABINA)
datasets involving patients from Europe and North
America.
ED, emergency department;
EOS, eosinophils; ICS,
inhaled corticosteroid; ITT, intention-to-treat; OLE,
open‑label
extension; Q2W, once every 2 weeks; SOC,
standard of care.
ED, emergency department; EOS, eosinophils; ICS, inhaled corticosteroid; ITT, intention-to-treat; OLE, open‑label extension; Q2W, once every 2 weeks; SOC, standard of care.